An accelerated article preview of “Selective silencing of euchromatic L1s revealed by genome-wide screens for L1 regulators” by Nian Liu, Cameron H. Lee, Tomek Swigut, Edward Grow, Bo Gu, Michael Bassik, and Joanna Wysocka is available online in Nature.
Liu N, Lee CH, Swigut T, Grow E, Gu B, Bassik M, Wysocka J. (2017) Nature. 2017 Dec 6. doi: 10.1038/nature25179.
29211708.
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The recent publication of “Transcription-Replication Conflict Orientation Modulates R-Loop Levels and Activates Distinct DNA Damage Responses” by Stephan Hamperl, Michael Bocek, Joshua Saldivar, Tomek Swigut, and Karlene Cimprich were featured in a Cell Preview and recommended on F1000Prime.
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“Antigen Presentation Profiling Reveals Recognition of Lymphoma Immunoglobulin Neoantigens” has been accepted for publication in Nature. Niclas Olsson (CSB, Elias Lab) is the co-first-author along with Michael Khodadoust (Oncology, Alizadeh Lab). Joshua Elias (CSB) is the co-corresponding author with Ash Alizadeh (Oncology).
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Julia Kamenz and Jim Ferrell have written a commentary on “Temporal Ordering of Cell Cycle Phosphorylation” that will be published on February 3rd, 2017 in Molecular Cell. Julia Kamenz has also been selected to give a talk on her work on this topic at the Winter qBio Meeting in Kaui from February 24th through February 27th, 2017.
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The recent publication from the lab of Daria Mochly-Rosen, “Potential biomarkers to follow the progression and treatment response of Huntington’s disease,” has been covered by Huntington’s Disease News and Silicon Republic.
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Prion proteins, best known as the agents of deadly brain disorders like mad cow disease, can help yeast survive hard times and pass the advantageous traits down to their offspring, according to a new study by researchers at the Stanford University School of Medicine.
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In the June 30th issue of Cell, Dr. Steven Cappell, working with his colleagues in the Meyer Lab, shows how diverse cellular stresses can over-ride mitogen signaling and send cells back to a quiescent state.
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The standard theory on negative cooperativity where the binding of one ligand makes it harder for a second ligand to bind to multi-subunit receptors says that the higher the negative cooperativity, the more graded the receptor’s response. It turns out that there is a little algebraic shortcut built into the standard theory, but in many situations, especially intracellular signaling, this assumption does not hold.
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CSB Professor Karlene Cimprich’s paper “Transcription-coupled nucleotide excision repair factors promote R-loop-induced genome instability” was recently recommended by the Faculty of 1000.
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The recent Science publication from the lab of Mary Teruel, “Controlling low rates of cell differentiation through noise and ultra-high feedback”, has been selected as a Science Editors’ Choice and has been featured in SCOPE, the Stanford School of Medicine online Blog.
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