Dear CSB Friends,
Join us! The Cimprich Lab will be hosting their lab happy hour this, Friday, July 19th starting at 4:30pm on Discovery Walk, close to Clark!
What: Cimprich Lab Happy Hour
When: Friday, July 19, 2024
Where: Discovery Walk (close to Clark)
Time: 4:30pm
We hope to see you there!
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The Cimprich Lab has a new lab website! To check out the newly designed website, please head over to http://cimprich.stanford.edu.
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Dr. Karlene Cimprich was awarded a BRCA Research Collaborative Grant from the V Foundation.
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An accelerated article preview of “Selective silencing of euchromatic L1s revealed by genome-wide screens for L1 regulators” by Nian Liu, Cameron H. Lee, Tomek Swigut, Edward Grow, Bo Gu, Michael Bassik, and Joanna Wysocka is available online in Nature.
Liu N, Lee CH, Swigut T, Grow E, Gu B, Bassik M, Wysocka J. (2017) Nature. 2017 Dec 6. doi: 10.1038/nature25179.
29211708.
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Postdocs Stephan Hamperl and Niclas Olsson will be representing CSB at the Breakthrough Prize Symposium Life Sciences Poster Session taking place at the Stanford Fisher Conference Center from 11:45 AM – 12:45 PM on Monday, December 4th.
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The recent publication of “Transcription-Replication Conflict Orientation Modulates R-Loop Levels and Activates Distinct DNA Damage Responses” by Stephan Hamperl, Michael Bocek, Joshua Saldivar, Tomek Swigut, and Karlene Cimprich were featured in a Cell Preview and recommended on F1000Prime.
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Professor Karlene Cimprich, postdoctoral researchers Stephan Hamperl, and Joshua Saldivar; graduate student Michael Bocek; and senior research scientist Tomek Swigut were featured in the Stanford Medicine blog SCOPE.
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Join us for the 1st Stanford Chromosome Dynamics and Genome Maintenance Symposium on Friday, March 31st from 9:15am-5:30pm. Click here for more details, and to register/present a poster by March 15th!
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Karlene has been elected as a fellow by the American Association for the Advancement of Science for her contributions to the understanding of genome maintenance, particularly for elucidating molecular mechanisms of DNA damage signaling and cellular sources of genome instability.
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